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Article title
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Abstract
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Keywords
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Introduction
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Pathogenesis of PD
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α-Synuclein: misfolding and aggregation
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Mitochondrial dysfunction
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Neuroinflammation
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Environmental factors
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Current therapeutic approaches
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DA-ergic approaches
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Non-DAergic approaches – mAChR inhibitors, NMDA blockers
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Multi-target treatment
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MAO and ChE
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MAO and antioxidants
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MAO-B & MAO-A inhibitors
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MAO inhib and H3 antag
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Miscellaneous
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Drawbacks of multi-target MAO-B inhibitor development
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Future perspectives
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Additional information
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References
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